Levodopa withdrawal and the neuroleptic malignant syndrome.
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منابع مشابه
Levodopa withdrawal and the neuroleptic malignant syndrome.
Levodopa withdrawal and the neuroleptic malignant syndrome Sir, In the paper: 'Levodopa withdrawal syndrome identical to neuroleptic-malignant syndrome' (Gibb & Griffith, 1986) the authors suggested that sudden withdrawal of dopaminergic drugs precipitated the development of neuroleptic-malignant syndrome (NMS) in their patient. However, it is difficult to reconcile the wide range of this patie...
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A 60 year old woman with idiopathic Parkinson's disease had been prescribed thioridazine for schizophrenia. Five months after this was stopped, Sinemet also considered of dubious therapeutic value, was withdrawn. One week later she developed features of the neuroleptic malignant syndrome (NMS) accompanied by myoglobinuric renal failure. Post-mortem examination confirmed Lewy body degeneration i...
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Neuroleptic malignant syndrome (NMS) is a rare but life-threatening idiosyncratic side effect resulting from neuroleptic drugs. NMS mainly occurs in patients treated with high-potency typical antipsychotics, but rarely caused by atypical antipsychotics. Although NMS is less common with atypical antipsychotic, but it seems that its incidence is rising due to increased administration of such drug...
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A patient with symptoms suggestive of neuroleptic malignant syndrome after levodopa withdrawal is described. The patient presented with persistent high fever, stupor, autonomic dysfunction, rigidity, and rhabdomyolysis. He was successfully treated with intravenous dantrolene, resumption of levodopa, and forced alkaline diuresis. Doctors should be aware of the risk of abrupt cessation of dopamin...
متن کاملNeuroleptic malignant syndrome.
The neuroleptic malignant syndrome is an uncommon, severe illness that consists of fever, muscular rigidity, and stupor. Various neuroleptics have been associated with the disease. A detailed neurological, medical, and neuropathological evaluation of this case was performed. Presumably, the syndrome is secondary to biochemical dysfunction of the basal ganglia and possible of the hypothalmus.
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ژورنال
عنوان ژورنال: Postgraduate Medical Journal
سال: 1986
ISSN: 0032-5473
DOI: 10.1136/pgmj.62.729.701